As one of the most common malignant tumors among women, the rapid growth and metastasis of breast cancer are inseparable from the remodeling of energy metabolism patterns. The potential mechanism underlying glucose metabolic reprogramming in breast cancer remains not fully elucidated. Recently, the research team led by Professor Gao Peng from the Department of Pathology, School of Basic Medical Sciences, Shandong University, has published their latest findings in the international journal Advanced Science (CAS Category 1, IF=14.1). The study is entitled “METTL14-mediated m6A modification of LINC01094 induces glucose metabolic reprogramming in breast cancer by recruiting the PKM2/JMJD5 complex”. This research reveals that the PKM2/JMJD5 complex, related to glucose metabolism, induces glucose metabolic reprogramming in breast cancer cells and promotes tumor progression.